Molecular biologists have eliminated some of the symptoms of Huntington's disease in mice.
To do this, scientists have normalized the work of the enzyme PRMT6, which plays an important role in the transmission of chemical signals to nerve endings.
According to one of the study's authors, Manuela Basson, an associate professor at the University of Trento, the enzyme PRMT6 marks molecules in Huntington's protein (a mutation that causes Huntington's disease). It has been found that these signals are important for the normal functioning of the protein chain involved in the transmission of signals within nerve cells.
Huntington's disease is a serious nervous system disease associated with a mutation in the HTT gene.